dc.contributor.author | Gokce, Hatice Humeyra Yavuz | |
dc.contributor.author | Dasdemir, Selcuk | |
dc.contributor.author | Kucukali, Cem Ismail | |
dc.contributor.author | Iplik, Elif Sinem | |
dc.contributor.author | Cakmakoglu, Bedia | |
dc.date.accessioned | 2021-12-21T08:45:47Z | |
dc.date.available | 2021-12-21T08:45:47Z | |
dc.date.issued | 2020 | |
dc.identifier.issn | 0101-6083 | |
dc.identifier.issn | 1806-938X | |
dc.identifier.uri | https://doi.org/10.1590/0101-60830000000227 | |
dc.identifier.uri | http://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1769 | |
dc.description.abstract | Background: Various studies demonstrating enhanced vulnerability to apoptosis may contribute to the pathobiology of schizophrenia. Objective: Thus, G proteins may provide an intriguing link between the signal transduction, and apoptotic hypotheses of schi | |
dc.description.sponsorship | Research Fund of Istanbul UniversityIstanbul University [11081] | |
dc.language.iso | English | |
dc.publisher | Unıv Sao Paulo, Inst Psıquıatrıa | |
dc.rights | gold | |
dc.title | G protein gene variants in schizophrenia | |
dc.type | Article | |
dc.relation.journal | Archıves Of Clınıcal Psychıatry | |
dc.identifier.issue | 2 | |
dc.identifier.startpage | 31 | |
dc.identifier.endpage | 34 | |
dc.identifier.volume | 47 | |
dc.identifier.doi | 10.1590/0101-60830000000227 | |
dc.relation.issue | 2 | |
dc.relation.volume | 47 | |