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dc.contributor.authorKocaturk, N.M.
dc.contributor.authorAkkoc, Y.
dc.contributor.authorKig, C.
dc.contributor.authorBayraktar, O.
dc.contributor.authorGozuacik, D.
dc.contributor.authorKutlu, O.
dc.date.accessioned2021-12-21T08:40:53Z
dc.date.available2021-12-21T08:40:53Z
dc.date.issued2019
dc.identifier.urihttps://doi.org/10.1016/j.ejps.2019.04.011
dc.identifier.urihttp://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1241
dc.description.abstractAutophagy is an evolutionarily conserved catabolic mechanism, by which eukaryotic cells recycle or degrades internal constituents through membrane-trafficking pathway. Thus, autophagy provides the cells with a sustainable source of biomolecules and energy
dc.description.sponsorshipThis work was supported by Scientific and Technological Research Council of Turkey (TUBITAK)-1001 Grant number: 114Z836 . YA is supported by TUBITAK-BIDEB 2211 Scholarships for his PhD studies.
dc.titleAutophagy as a molecular target for cancer treatment
dc.relation.journalEuropean Journal of Pharmaceutical Sciences
dc.identifier.startpage116
dc.identifier.endpage137
dc.identifier.volume134
dc.identifier.doi10.1016/j.ejps.2019.04.011
dc.relation.volume134


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