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dc.contributor.authorPekmezci, M.
dc.contributor.authorPhillips, J.J.
dc.contributor.authorDirilenoglu, F.
dc.contributor.authorAtasever-Rezanko, T.
dc.contributor.authorTihan, T.
dc.contributor.authorSolomon, D.
dc.contributor.authorBollen, A.
dc.contributor.authorPerry, A.
dc.date.accessioned2021-12-21T08:40:47Z
dc.date.available2021-12-21T08:40:47Z
dc.date.issued2020
dc.identifier.issn16322
dc.identifier.urihttps://doi.org/10.1007/s00401-019-02123-8
dc.identifier.urihttp://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1191
dc.description.abstract[No abstract available]
dc.description.sponsorshipThis study was funded in part by the UCSF Department of Pathology?(M.P.), NIH/NCI U01 (CA229345, J.J.P.) and NIH Director?s Early Independence Award (DP5 OD021403, D.A.S.).
dc.language.isoEnglish
dc.publisherSpringer
dc.titleLoss of H3K27 trimethylation by immunohistochemistry is frequent in oligodendroglioma, IDH-mutant and 1p/19q-codeleted, but is neither a sensitive nor a specific marker
dc.typeLetter
dc.relation.journalActa Neuropathologica
dc.identifier.issue3
dc.identifier.startpage597
dc.identifier.endpage600
dc.identifier.volume139
dc.identifier.doi10.1007/s00401-019-02123-8
dc.relation.issue3
dc.relation.volume139


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