dc.contributor.author | Gokce, H.H.Y. | |
dc.contributor.author | Dasdemir, S. | |
dc.contributor.author | Kucukali, C.I. | |
dc.contributor.author | Iplik, E.S. | |
dc.contributor.author | Cakmakoglu, B. | |
dc.date.accessioned | 2021-12-21T08:40:46Z | |
dc.date.available | 2021-12-21T08:40:46Z | |
dc.date.issued | 2020 | |
dc.identifier.issn | 1016083 | |
dc.identifier.uri | https://doi.org/10.1590/0101-60830000000227 | |
dc.identifier.uri | http://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1188 | |
dc.description.abstract | Background: Various studies demonstrating enhanced vulnerability to apoptosis may contribute to the pathobiology of schizophrenia. Objective: Thus, G proteins may provide an intriguing link between the signal transduction, and apoptotic hypotheses of schi | |
dc.description.sponsorship | This work was supported by the Research Fund of Istanbul University. (Project No: 11081). | |
dc.language.iso | English | |
dc.publisher | Universidade de Sao Paulo | |
dc.rights | All Open Access, Gold | |
dc.title | G protein gene variants in schizophrenia | |
dc.type | Article | |
dc.relation.journal | Revista de Psiquiatria Clinica | |
dc.identifier.issue | 2 | |
dc.identifier.startpage | 31 | |
dc.identifier.endpage | 34 | |
dc.identifier.volume | 47 | |
dc.identifier.doi | 10.1590/0101-60830000000227 | |
dc.relation.issue | 2 | |
dc.relation.volume | 47 | |