Combined evaluation of proliferation and apoptosis to calculate IC50 of VPA-induced PANC-1 cells and assessing its effect on the Wnt signaling pathway

dc.contributor.author	Ekici, Y.
dc.contributor.author	Yilmaz, A.
dc.contributor.author	Kucuksezer, U.C.
dc.contributor.author	Gazioglu, S.B.
dc.contributor.author	Yamalioglu, Z.D.
dc.contributor.author	Gurol, A.O.
dc.contributor.author	Linn, T.
dc.contributor.author	Tuncer, F.N.
dc.date.accessioned	2021-12-21T08:40:29Z
dc.date.available	2021-12-21T08:40:29Z
dc.date.issued	2021
dc.identifier.issn	13570560
dc.identifier.uri	https://doi.org/10.1007/s12032-021-01560-4
dc.identifier.uri	http://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1013
dc.description.abstract	Pancreatic ductal adenocarcinoma (PDAC) is among the most deadly cancers. Since most patients develop resistance to conventional treatments, new approaches are in urgency. Valproic acid (VPA) was shown to induce apoptosis and reduce proliferation in PANC-
dc.description.sponsorship	This work was supported by the Scientific Research Projects Coordination Unit of Istanbul University. Project No.: 32763.
dc.language.iso	English
dc.publisher	Springer
dc.title	Combined evaluation of proliferation and apoptosis to calculate IC50 of VPA-induced PANC-1 cells and assessing its effect on the Wnt signaling pathway
dc.type	Article
dc.relation.journal	Medical Oncology
dc.identifier.issue	9
dc.identifier.volume	38
dc.identifier.doi	10.1007/s12032-021-01560-4
dc.relation.issue	9
dc.relation.volume	38