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dc.contributor.authorDemiryas, S.
dc.contributor.authorCaliskan, R.
dc.contributor.authorSaribas, S.
dc.contributor.authorAkkus, S.
dc.contributor.authorGareayaghi, N.
dc.contributor.authorKirmusaoglu, S.
dc.contributor.authorKepil, N.
dc.contributor.authorDinc, H.
dc.contributor.authorDag, H.
dc.contributor.authorDagdeviren, E.
dc.contributor.authorTokman, H. B.
dc.date.accessioned2021-12-21T08:47:40Z
dc.date.available2021-12-21T08:47:40Z
dc.date.issued2020
dc.identifier.issn1784-3227
dc.identifier.urihttp://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/2283
dc.description.abstractIntroduction : As a component of the cag T4SS, the cagL gene is invoked in the translocation of CagA into host cells and is essential for the formation of cag PAI-associated pili between H. pylori and gastric epithelial cells. Aim : We aimed to investigat
dc.description.sponsorshipIstanbul University Research FundIstanbul University [45151]
dc.language.isoEnglish
dc.publisherUnıv Catholıque Louvaın-Ucl
dc.titleThe association between cagL and cagA, vacAs/m, babA genes in patients with gastric cancer, duodenal ulcer, and non-ulcer dyspepsia related to Helicobacter pylori
dc.typeArticle
dc.relation.journalActa Gastro-Enterologıca Belgıca
dc.identifier.issue3
dc.identifier.startpage385
dc.identifier.endpage392
dc.identifier.volume83
dc.relation.issue3
dc.relation.volume83


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