Combined evaluation of proliferation and apoptosis to calculate IC50 of VPA-induced PANC-1 cells and assessing its effect on the Wnt signaling pathway

dc.contributor.author	Ekici, Yeliz
dc.contributor.author	Yilmaz, Abdullah
dc.contributor.author	Kucuksezer, Umut Can
dc.contributor.author	Gazioglu, Sema Bilgic
dc.contributor.author	Yamalioglu, Zeynep Dogusan
dc.contributor.author	Gurol, Ali Osman
dc.contributor.author	Linn, Thomas
dc.contributor.author	Tuncer, Feyza Nur
dc.date.accessioned	2021-12-21T08:46:52Z
dc.date.available	2021-12-21T08:46:52Z
dc.date.issued	2021
dc.identifier.issn	1357-0560
dc.identifier.issn	1559-131X
dc.identifier.uri	https://doi.org/10.1007/s12032-021-01560-4
dc.identifier.uri	http://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/2115
dc.description.abstract	Pancreatic ductal adenocarcinoma (PDAC) is among the most deadly cancers. Since most patients develop resistance to conventional treatments, new approaches are in urgency. Valproic acid (VPA) was shown to induce apoptosis and reduce proliferation in PANC-
dc.description.sponsorship	Scientific Research Projects Coordination Unit of Istanbul UniversityIstanbul University [32763]
dc.language.iso	English
dc.publisher	Humana Press Inc
dc.title	Combined evaluation of proliferation and apoptosis to calculate IC50 of VPA-induced PANC-1 cells and assessing its effect on the Wnt signaling pathway
dc.type	Article
dc.relation.journal	Medıcal Oncology
dc.identifier.issue	9
dc.identifier.volume	38
dc.identifier.doi	10.1007/s12032-021-01560-4
dc.relation.issue	9
dc.relation.volume	38

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