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dc.contributor.authorKuran, S.B.
dc.contributor.authorIplik, E.S.
dc.contributor.authorCakmakoglu, B.
dc.contributor.authorKahraman, O.T.
dc.contributor.authorIyibozkurt, A.C.
dc.contributor.authorKoc, A.
dc.contributor.authorErgen, A.
dc.contributor.authorYilmaz, S.G.
dc.contributor.authorIsbir, T.
dc.date.accessioned2021-12-21T08:41:10Z
dc.date.available2021-12-21T08:41:10Z
dc.date.issued2018
dc.identifier.issn1455680
dc.identifier.urihttps://doi.org/10.14715/cmb/2018.64.4.13
dc.identifier.urihttp://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1342
dc.description.abstractReactive oxygen species (ROS) have been shown to be responsible for inducing DNA damage leading to mutagenesis, carcinogenesis, and cell death if the capacity of the protective antioxidant system is impaired. Endometrial carcinoma is the primary cancer ty
dc.language.isoEnglish
dc.publisherCellular and Molecular Biology Association
dc.titleRelation of MPO, MnSOD, NQO1 gene variants in endometrial carcinoma in the line of PCR-RFLP methods
dc.typeArticle
dc.relation.journalCellular and Molecular Biology
dc.identifier.issue4
dc.identifier.startpage78
dc.identifier.endpage82
dc.identifier.volume64
dc.identifier.doi10.14715/cmb/2018.64.4.13
dc.relation.issue4
dc.relation.volume64


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