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dc.contributor.authorÇalışkan, Z.
dc.contributor.authorMutlu, T.
dc.contributor.authorGüven, M.
dc.contributor.authorTunçdemir, M.
dc.contributor.authorNiyazioğlu, M.
dc.contributor.authorHacioglu, Y.
dc.contributor.authorDincer, Y.
dc.date.accessioned2021-12-21T08:41:06Z
dc.date.available2021-12-21T08:41:06Z
dc.date.issued2018
dc.identifier.issn3781119
dc.identifier.urihttps://doi.org/10.1016/j.gene.2017.11.071
dc.identifier.urihttp://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1320
dc.description.abstractSirtuins (SIRTs) is a family of NAD+ dependent histone deacetylases. SIRT6 takes play in glucose homeostasis, genomic stability and DNA repair. Although increased oxidative DNA damage and decreased DNA repair activity were determined in diabetes mellitus,
dc.description.sponsorshipThis work was supported and funded by Istanbul University Scientific Research Projects Unit (Project No: 53685 and BYP-2017-27116) and by TUBITAK 2211-C Domestic Graduate Scholarship Program.
dc.language.isoEnglish
dc.publisherElsevier B.V.
dc.titleSIRT6 expression and oxidative DNA damage in individuals with prediabetes and type 2 diabetes mellitus
dc.typeArticle
dc.relation.journalGene
dc.identifier.startpage542
dc.identifier.endpage548
dc.identifier.volume642
dc.identifier.doi10.1016/j.gene.2017.11.071
dc.relation.volume642


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