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dc.contributor.authorGungor-Ordueri, N.E.
dc.contributor.authorKuscu, N.
dc.contributor.authorTasatargil, A.
dc.contributor.authorBurgucu, D.
dc.contributor.authorKaracan, M.
dc.contributor.authorCelik-Ozenci, C.
dc.date.accessioned2021-12-21T08:40:53Z
dc.date.available2021-12-21T08:40:53Z
dc.date.issued2019
dc.identifier.issn17341140
dc.identifier.urihttps://doi.org/10.1016/j.pharep.2019.02.018
dc.identifier.urihttp://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1237
dc.description.abstractBackground: Doxorubicin (DOX), is a chemotherapeutic agent, which evokes oxidative stress and cell apoptosis in testicular tissue. It is known that the activation of the nuclear enzyme poly (ADP-ribose) polymerase (PARP), leading to apoptosis induced by D
dc.description.sponsorshipTurkish Scientific and Research Council (TUBITAK) supported this work with project number of 110S377 . This work was partially supported by the Akdeniz University Research Foundation, Antalya, Turkey .
dc.language.isoEnglish
dc.publisherElsevier B.V.
dc.titleDoxorubicin-induced testicular damage is related to PARP-1 signaling molecules in mice
dc.typeArticle
dc.relation.journalPharmacological Reports
dc.identifier.issue4
dc.identifier.startpage591
dc.identifier.endpage602
dc.identifier.volume71
dc.identifier.doi10.1016/j.pharep.2019.02.018
dc.relation.issue4
dc.relation.volume71


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