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dc.contributor.authorGokce, H.H.Y.
dc.contributor.authorDasdemir, S.
dc.contributor.authorKucukali, C.I.
dc.contributor.authorIplik, E.S.
dc.contributor.authorCakmakoglu, B.
dc.date.accessioned2021-12-21T08:40:46Z
dc.date.available2021-12-21T08:40:46Z
dc.date.issued2020
dc.identifier.issn1016083
dc.identifier.urihttps://doi.org/10.1590/0101-60830000000227
dc.identifier.urihttp://dspace.yeniyuzyil.edu.tr:8080/xmlui/handle/20.500.12629/1188
dc.description.abstractBackground: Various studies demonstrating enhanced vulnerability to apoptosis may contribute to the pathobiology of schizophrenia. Objective: Thus, G proteins may provide an intriguing link between the signal transduction, and apoptotic hypotheses of schi
dc.description.sponsorshipThis work was supported by the Research Fund of Istanbul University. (Project No: 11081).
dc.language.isoEnglish
dc.publisherUniversidade de Sao Paulo
dc.rightsAll Open Access, Gold
dc.titleG protein gene variants in schizophrenia
dc.typeArticle
dc.relation.journalRevista de Psiquiatria Clinica
dc.identifier.issue2
dc.identifier.startpage31
dc.identifier.endpage34
dc.identifier.volume47
dc.identifier.doi10.1590/0101-60830000000227
dc.relation.issue2
dc.relation.volume47


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